Chronic tooth pulp inflammation induces persistent expression of phosphorylated ERK (pERK) and phosphorylated p38 (pp38) in trigeminal subnucleus caudalis

Author:

Worsley M.A.,Allen C.E.,Billinton A.,King A.E.,Boissonade F.M.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference45 articles.

1. Inflammation persistently enhances nocifensive behaviors mediated by spinal group I mGluRs through sustained ERK activation;Adwanikar;Pain,2004

2. Barber MW, Loescher AR, Boissonade FM, Rodd HD (2012), Mechanisms of thermal sensitivity and pulpal pain: clinical findings from quantitative thermal testing [Abstract PF 350]. In: IASP 14th World Congress on Pain), Milan.

3. Evidence for anti-inflammatory and putative analgesic effects of a monoclonal antibody to calcitonin gene-related peptide;Bowler;Neuroscience,2013

4. Regulation of peripheral inflammation by spinal p38 MAP kinase in rats;Boyle;PLoS Med,2006

5. Chronic tooth pulp inflammation causes transient and persistent expression of Fos in dynorphin-rich regions of rat brainstem;Byers;Brain Res,2000

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