Chronic myeloid leukemia and BCR/ABL signal pathways are not associated with AKT1 pleckstrin homology domain (E17K) mutations
Author:
Publisher
Wiley
Subject
Hematology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0609.2009.01350.x/fullpdf
Reference13 articles.
1. The phosphatidyl inositol 3-Kinase AKT pathway in human cancer;Vivanco;Nat Rev Cancer,2002
2. AKT/PKB signaling: navigating downstream;Manning;Cell,2007
3. Rare PIK3CA hotspot mutations in carcinomas of the biliary tract;Riener;Genes Chromosomes Cancer,2008
4. A transforming mutation in the pleckstrin homology domain of AKT1 in cancer;Carpten;Nature,2007
5. Mutational analysis of oncogenic AKT E17K mutation in common solid cancers and acute leukaemias;Kim;Br J Cancer,2008
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1. Differential Effects of Selective Inhibitors Targeting the PI3K/AKT/mTOR Pathway in Acute Lymphoblastic Leukemia;PLoS ONE;2013-11-14
2. The PI3K/PKB signaling module as key regulator of hematopoiesis: implications for therapeutic strategies in leukemia;Blood;2012-01-26
3. Reversible phosphorylation in haematological malignancies: Potential role for protein tyrosine phosphatases in treatment?;Biochimica et Biophysica Acta (BBA) - Reviews on Cancer;2010-12
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