Selective Translation of Mengovirus RNA over Host mRNA in Homologous, Fractionated, Cell-Free Translational Systems from Ehrlich-Ascites-Tumor Cells
Author:
Publisher
Wiley
Subject
Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1432-1033.1978.tb12101.x/fullpdf
Reference25 articles.
1. 1. D. Baltimore, and H. B. Levy (1969 ) inThe Biochemistry of Viruses () pp.101 -176 , Marcel Dekker, New York.
2. Fate of mRNA of L-Cells Infected with Mengovirus
3. Encephalomyocarditis Virus Infection of Mouse Plasmacytoma Cells I. Inhibition of Cellular Protein Synthesis
4. Poliovirus Infection and Poly(A) Sequences of Cytoplasmic Cellular RNA
5. Structural difference between the 5' termini of viral and cellular mRNA in poliovirus-infected cells: possible basis for the inhibition of host protein synthesis
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1. Translation of Encephalomyocarditis Virus RNA by Internal Ribosomal Entry;Current Topics in Microbiology and Immunology;1995
2. Initiation of protein synthesis by internal entry of ribosomes into the 5' nontranslated region of encephalomyocarditis virus RNA in vivo;Journal of Virology;1989-04
3. Picornaviruses at the Molecular Level;The Molecular Basis of Viral Replication;1987
4. Evidence for the presence of an inhibitor on ribosomes in mouse L cells infected with mengovirus;Journal of Virology;1985-10
5. A Direct Correlation Between the Affinity of a Given mRNa for Eukaryotic Initiation Factor 2 and its Ability to Compete in Translation;Proceedings in Life Sciences;1984
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