Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms
Author:
Publisher
Hindawi Limited
Subject
Virology,Immunology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1462-5822.2006.00834.x/fullpdf
Reference36 articles.
1. Structural Alteration of the Membrane of Erythrocytes Infected with Babesia bovis
2. Knob-positive and knob-negative Plasmodium falciparum differ in expression of a strain-specific malarial antigen on the surface of infected erythrocytes.
3. Plasmodium falciparum likely encodes the principal anion channel on infected human erythrocytes
4. The ves Multigene Family of B. bovis Encodes Components of Rapid Antigenic Variation at the Infected Erythrocyte Surface
5. Cloning the P. falciparum gene encoding PfEMP1, a malarial variant antigen and adherence receptor on the surface of parasitized human erythrocytes
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