Activation of the mTOR/HIF‐1α/VEGF axis promotes M1 macrophage polarization in non‐eosinophilic chronic rhinosinusitis with nasal polyps-Reference-Cited by-同舟云学术

Activation of the mTOR/HIF‐1α/VEGF axis promotes M1 macrophage polarization in non‐eosinophilic chronic rhinosinusitis with nasal polyps

Published:2021-08-21 Issue:2 Volume:77 Page:643-646
ISSN:0105-4538
Container-title:Allergy
language:en
Short-container-title:Allergy

Author:

Zhong Bing¹,Du Jintao¹,Liu Feng¹,Liu Yafeng¹,Liu Shixi¹,Xie Lifeng²,Wang De yun³^ORCID,Ba Luo²^ORCID

Affiliation:

1. Upper Airways Research Laboratory Department of Otolaryngology—Head and Neck Surgery West China Hospital, Sichuan University Chengdu China

2. Department of Otolaryngology People's Hospital of Tibet Autonomous Region Lhasa China

3. Department of Otolaryngology Yong Loo Lin School of Medicine National University of Singapore, National University Health System Singapore Singapore

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/all.15050

Reference6 articles.

1. Hypoxia‐induced factor‐1α induces NLRP3 expression by M1 macrophages in noneosinophilic chronic rhinosinusitis with nasal polyps

2. Hypoxia: Turning vessels into vassals of cancer immunotolerance

3. Mammalian target of rapamycin regulates neutrophil extracellular trap formation via induction of hypoxia-inducible factor 1 α

4. TSC2 regulates VEGF through mTOR-dependent and -independent pathways

5. Inflammation and Hypoxia: HIF and PHD Isoform Selectivity

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