Influence of CIT‐Induced DGF on Kidney Transplant Outcomes
Author:
Affiliation:
1. Montefiore Medical Center, Bronx, NY
2. Glickman Urological and Kidney Institute
3. Department of Quantitative Health Sciences, Cleveland Clinic, Cleveland, OH
Publisher
Wiley
Subject
Pharmacology (medical),Transplantation,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1600-6143.2011.03817.x
Reference27 articles.
1. The ‘Two, One, Zero’ Decision: What to Do with Suboptimal Deceased Donor Kidneys
2. Improving Distribution Efficiency of Hard-to-Place Deceased Donor Kidneys: Predicting Probability of Discard or Delay
3. Possible role of postischemic reperfusion injury as initiator of allorecognition/alloactivation;Land W.;Transplant Proc,1998
4. ANTIOXIDANT LAZAROID U-74006F IMPROVES RENAL FUNCTION AND REDUCES THE EXPRESSION OF CYTOKINES, INDUCIBLE NITRIC OXIDE SYNTHASE, AND MHC ANTIGENS IN A SYNGENEIC RENAL TRANSPLANT MODEL
5. The “injury response”: A concept linking nonspecific injury, acute rejection, and long-term transplant outcomes
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