Amyotrophic lateral sclerosis‐associated Vap33 is required for maintaining neuronal dendrite morphology and organelle distribution in Drosophila
Author:
Affiliation:
1. Program of Biomedical Science and Basic Biology Graduate School of Integrated Sciences for Life Hiroshima University Hiroshima Japan
2. Department of Genetics Graduate School of Pharmaceutical Sciences The University of Tokyo Tokyo Japan
Funder
Japan Society for the Promotion of Science
Publisher
Wiley
Subject
Cell Biology,Genetics
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/gtc.12835
Reference34 articles.
1. Amyotrophic lateral sclerosis-related VAPB P56S mutation differentially affects the function and survival of corticospinal and spinal motor neurons
2. Protein Folding and Quality Control in the ER
3. hVAPB, the causative gene of a heterogeneous group of motor neuron diseases in humans, is functionally interchangeable with its Drosophila homologue DVAP-33A at the neuromuscular junction
4. Characterization of the Properties of a Novel Mutation in VAPB in Familial Amyotrophic Lateral Sclerosis
5. Cytoplasmic and mitochondrial protein translation in axonal and dendritic terminal arborization
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