Improved platelet counts during prolonged tranexamic therapy for Quebec platelet disorder implicate the underlying fibrinolytic defect as the cause of lower platelet counts-Reference-Cited by-同舟云学术

Improved platelet counts during prolonged tranexamic therapy for Quebec platelet disorder implicate the underlying fibrinolytic defect as the cause of lower platelet counts

Published:2020-08-09 Issue:6 Volume:42 Page:
ISSN:1751-5521
Container-title:International Journal of Laboratory Hematology
language:en
Short-container-title:Int J Lab Hem

Author:

Hayward Catherine P.M.¹²³^ORCID,Tasneem Subia¹^ORCID,Rivard Georges E.³⁴

Affiliation:

1. Department of Pathology and Molecular Medicine McMaster University Hamilton ON Canada

2. Department of Medicine McMaster University Hamilton ON Canada

3. Hamilton Regional Laboratory Medicine Program Hamilton ON Canada

4. Centre Hospitalier Universitaire Sainte Justine Montreal QC Canada

Funder

Canadian Institutes of Health Research

Publisher

Wiley

Subject

Biochemistry (medical),Clinical Biochemistry,Hematology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/ijlh.13311

Reference12 articles.

1. The genetics of platelet count and volume in humans

2. Thrombopoietin levels in Quebec platelet disorder-Implications for the mechanism of thrombocytopenia

3. Persons with Quebec platelet disorder have a tandem duplication of PLAU, the urokinase plasminogen activator gene

4. The duplication mutation of Quebec platelet disorder dysregulates PLAU, but not C10orf55, selectively increasing production of normal PLAU transcripts by megakaryocytes but not granulocytes

5. Platelets from patients with the Quebec platelet disorder contain and secrete abnormal amounts of urokinase-type plasminogen activator

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