ATF3 represses PINK1 gene transcription in lung epithelial cells to control mitochondrial homeostasis-Reference-Cited by-同舟云学术

ATF3 represses PINK1 gene transcription in lung epithelial cells to control mitochondrial homeostasis

Published:2018-01-24 Issue:2 Volume:17 Page:e12720
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Bueno Marta¹²,Brands Judith¹²,Voltz Lauren¹,Fiedler Kaitlin¹,Mays Brenton¹,St. Croix Claudette³,Sembrat John¹⁴,Mallampalli Rama K.²⁵,Rojas Mauricio¹⁴,Mora Ana L.¹²

Affiliation:

1. Vascular Medicine Institute; Department of Medicine; University of Pittsburgh; Pittsburgh PA USA

2. Division of Pulmonary Allergy and Critical Care Medicine; Department of Medicine; University of Pittsburgh; Pittsburgh PA USA

3. Center for Biologic Imaging; University of Pittsburgh; Pittsburgh PA USA

4. The Dorothy P. and Richard P. Simmons Center for Interstitial Lung Diseases; University of Pittsburgh; Pittsburgh PA USA

5. Veterans Affairs Pittsburgh Healthcare System; Pittsburgh PA USA

Funder

National Institutes of Health

University of Pittsburgh

Institute for Transfusion Medicine

Hemophilia Center of Western Pennsylvania

U.S. Department of Veterans Affairs

Veterans Health Administration

Office of Research and Development

Biomedical Laboratory Research and Development

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12720/fullpdf

Reference37 articles.

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3. Global expression profiling of fibroblast responses to transforming growth factor-β1 reveals the induction of inhibitor of differentiation-1 and provides evidence of smooth muscle cell phenotypic switching;Chambers;American Journal of Pathology,2003

4. Analysis of ATF3, a transcription factor induced by physiological stresses and modulated by gadd153/Chop10;Chen;Molecular and Cellular Biology,1996

5. Upregulation of human PINK1 gene expression by NFkappaB signalling;Duan;Molecular Brain,2014

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