Lipid Deficiency Contributes to Impaired Alveolar Progenitor Cell Function in Aging and Idiopathic Pulmonary Fibrosis
Author:
Affiliation:
1. Department of Medicine and Women’s Guild Lung Institute, and
2. Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, California
Funder
National Heart, Lung, and Blood Institute
National Institute on Aging
Publisher
American Thoracic Society
Link
https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2023-0290OC
Reference53 articles.
1. Pulmonary fibrosis: patterns and perpetrators
2. Grp78 Loss in Epithelial Progenitors Reveals an Age-linked Role for Endoplasmic Reticulum Stress in Pulmonary Fibrosis
3. Hyaluronan and TLR4 promote surfactant-protein-C-positive alveolar progenitor cell renewal and prevent severe pulmonary fibrosis in mice
4. Single-cell RNA sequencing identifies diverse roles of epithelial cells in idiopathic pulmonary fibrosis
5. Contributions of alveolar epithelial cell quality control to pulmonary fibrosis
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