Grp78 Loss in Epithelial Progenitors Reveals an Age-linked Role for Endoplasmic Reticulum Stress in Pulmonary Fibrosis-Reference-Cited by-同舟云学术

Grp78 Loss in Epithelial Progenitors Reveals an Age-linked Role for Endoplasmic Reticulum Stress in Pulmonary Fibrosis

Published:2020-01-15 Issue:2 Volume:201 Page:198-211
ISSN:1073-449X
Container-title:American Journal of Respiratory and Critical Care Medicine
language:en
Short-container-title:Am J Respir Crit Care Med

Author:

Borok Zea¹²³⁴^ORCID,Horie Masafumi¹²,Flodby Per¹²,Wang Hongjun¹²,Liu Yixin¹²,Ganesh Sivagini¹²,Firth Amy L.¹²⁵,Minoo Parviz²⁶,Li Changgong⁶,Beers Michael F.⁷,Lee Amy S.³⁴,Zhou Beiyun¹²⁴^ORCID

Affiliation:

1. Division of Pulmonary, Critical Care and Sleep Medicine

2. Hastings Center for Pulmonary Research, Department of Medicine

3. Department of Biochemistry and Molecular Medicine

4. Norris Comprehensive Cancer Center

5. Department of Stem Cell Biology and Regenerative Medicine, and

6. Department of Pediatrics, Keck School of Medicine, University of Southern California, Los Angeles, California; and

7. Pulmonary, Allergy, and Critical Care Division of the University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania

Publisher

American Thoracic Society

Subject

Critical Care and Intensive Care Medicine,Pulmonary and Respiratory Medicine

Link

https://www.atsjournals.org/doi/pdf/10.1164/rccm.201902-0451OC

Reference49 articles.

1. The critical roles of endoplasmic reticulum chaperones and unfolded protein response in tumorigenesis and anticancer therapies

2. Role of the unfolded protein response, GRP78 and GRP94 in organ homeostasis

3. Role of Epithelial Cells in Idiopathic Pulmonary Fibrosis: From Innocent Targets to Serial Killers

4. Is Progression of Pulmonary Fibrosis due to Ventilation-induced Lung Injury?

5. Revealing the Pathogenic and Aging-related Mechanisms of the Enigmatic Idiopathic Pulmonary Fibrosis. An Integral Model

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