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ALK,ROS1andNTRK3gene rearrangements in inflammatory myofibroblastic tumours

  • Published:2016-01-19 Issue:1 Volume:69 Page:72-83
  • ISSN:0309-0167
  • Container-title:Histopathology
  • language:en
  • Short-container-title:Histopathology

Author:

Yamamoto Hidetaka1,Yoshida Akihiko2,Taguchi Kenichi3,Kohashi Kenichi1,Hatanaka Yui1,Yamashita Atsushi4,Mori Daisuke5,Oda Yoshinao1

Affiliation:

1. Department of Anatomical Pathology; Graduate School of Medical Sciences; Kyushu University; Fukuoka Japan

2. Department of Pathology and Clinical Laboratories; National Cancer Centre Hospital; Tokyo Japan

3. Department of Pathology; National Kyushu Cancer Center; Fukuoka Japan

4. Department of Pathology; Faculty of Medicine; University of Miyazaki; Kiyotake Japan

5. Department of Pathology; Saga Medical Centre Koseikan; Saga Japan

Funder

Japan Society for the Promotion of Science London

Publisher

Wiley

Subject

General Medicine,Histology,Pathology and Forensic Medicine

Reference41 articles.

1. Inflammatory myofibroblastic tumours: where are we now?;Gleason;J. Clin. Pathol.,2008

2. CD30 + anaplastic large cell lymphoma: a review of its histopathologic, genetic, and clinical features;Stein;Blood,2000

3. Identification of the transforming EML4-ALK fusion gene in non-small-cell lung cancer;Soda;Nature,2007

4. KIF5B-ALK, a novel fusion oncokinase identified by an immunohistochemistry-based diagnostic system for ALK-positive lung cancer;Takeuchi;Clin. Cancer Res.,2009

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