DNA damage in normally and prematurely aged mice-Reference-Cited by-同舟云学术

DNA damage in normally and prematurely aged mice

Published:2013-04-24 Issue:3 Volume:12 Page:467-477
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Maslov Alexander Y.¹,Ganapathi Shireen¹,Westerhof Maaike¹,Quispe‐Tintaya Wilber¹,White Ryan R.¹,Van Houten Bennett²,Reiling Erwin³⁴,Dollé Martijn E. T.³,Steeg Harry³,Hasty Paul⁵,Hoeijmakers Jan H. J.⁴,Vijg Jan¹

Affiliation:

1. Department of Genetics Albert Einstein College of Medicine New York NY 10461USA

2. Department of Pharmacology and Chemical Biology University of Pittsburgh Cancer Institute University of Pittsburgh School of Medicine Pittsburgh PA 15213USA

3. National Institute of Public Health and the Environment Bilthoven The Netherlands

4. MGC Department of Genetics CBG Cancer Genomics Center Erasmus Medical Center Rotterdam The Netherlands

5. Department of Molecular Medicine and Institute of Biotechnology University of Texas Health Science Center San Antonio TX 78245USA

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.12071

Reference36 articles.

1. Mitochondrial DNA damage is a hallmark of chemically induced and the R6/2 transgenic model of Huntington's disease

2. Measurement of oxidatively induced base lesions in liver from Wistar rats of different ages

3. The Free Radical Theory of Aging Matures

4. DNA repair in an active gene: Removal of pyrimidine dimers from the DHFR gene of CHO cells is much more efficient than in the genome overall

5. Accelerated Age-Related Cognitive Decline and Neurodegeneration, Caused by Deficient DNA Repair

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