Targeting the NLRP3 inflammasome to reduce warm ischemic injury in donation after circulatory death heart-Reference-Cited by-同舟云学术

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Targeting the NLRP3 inflammasome to reduce warm ischemic injury in donation after circulatory death heart

Published:2020-08-02 Issue:10 Volume:34 Page:
ISSN:0902-0063
Container-title:Clinical Transplantation
language:en
Short-container-title:Clin Transplant

Author:

Quader Mohammed¹^ORCID,Mezzaroma Eleonora²,Kenning Kristine¹,Toldo Stefano³^ORCID

Affiliation:

1. Division of Thoracic and Cardiovascular Surgery Pauley Heart Center Virginia Commonwealth University Richmond Virginia USA

2. School of Pharmacy Virginia Commonwealth University Richmond Virginia USA

3. Division of Cardiology Pauley Heart Center Virginia Commonwealth University Richmond Virginia USA

Funder

American Heart Association

Health Services Research and Development

Publisher

Wiley

Subject

Transplantation

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/ctr.14044

Reference30 articles.

1. Heart disease and stroke statistics–2015 update: a report from the American Heart Association;Mozaffarian D;Circulation,2015

2. Risk Assessment and Comparative Effectiveness of Left Ventricular Assist Device and Medical Management in Ambulatory Heart Failure Patients

3. OPTN/SRTR 2016 Annual Data Report: Heart

4. Outcomes of Donation After Circulatory Death Heart Transplantation in Australia

Cited by 14 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Heart Transplant and Donors After Circulatory Death: A Clinical-Preclinical Systematic Review;Journal of Surgical Research;2023-12

2. Outcomes of Heart Transplant Donation After Circulatory Death;Journal of the American College of Cardiology;2023-10

3. Baricitinib with cyclosporine eliminates acute graft rejection in fully mismatched skin and heart transplant models;Frontiers in Immunology;2023-09-06

4. Thoracic organ donation after circulatory determination of death;Transplantation Reports;2023-02

5. Influence of sex on global myocardial ischemia tolerance and mitochondrial function in circulatory death donor hearts;American Journal of Physiology-Heart and Circulatory Physiology;2023-01-01

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