Regulation of Toll-like receptor 2 interaction with Ecgp96 controlsEscherichia coli K1 invasion of brain endothelial cells

Author:

Krishnan Subramanian1,Chen Shuang2,Turcatel Gianluca3,Arditi Moshe2,Prasadarao Nemani V.14

Affiliation:

1. Division of Infectious Diseases; Children's Hospital Los Angeles; Los Angeles CA 90027 USA

2. Department of Pediatric Infectious Diseases and Immunology; Cedars-Sinai Medical Center; Los Angeles CA 90048 USA

3. Developmental Biology and Regenerative Medicine; Children's Hospital Los Angeles; Los Angeles CA 90027 USA

4. Department of Pediatrics; The Saban Research Institute; Children's Hospital of Los Angeles and Keck School of Medicine; University of Southern California; Los Angeles CA 90027 USA

Publisher

Hindawi Limited

Subject

Virology,Immunology,Microbiology

Reference43 articles.

1. An 11-year study of causes of neonatal bacterial meningitis in Ahvaz, Iran;Aletayeb;Pediatr Int,2010

2. Mice that exclusively express TLR4 on endothelial cells can efficiently clear a lethal systemic Gram-negative bacterial infection;Andonegui;J Clin Invest,2009

3. Evidence that glycoprotein 96 (B2), a stress protein, functions as a Th2-specific costimulatory molecule;Banerjee;J Immunol,2002

4. Gp96 is a receptor for a novel Listeria monocytogenes virulence factor, Vip, a surface protein;Cabanes;EMBO J,2005

5. Nuclear factor-kappaB: activation and regulation during Toll-like receptor signaling;Carmody;Cell Mol Immunol,2007

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