GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection-Reference-Cited by-同舟云学术

GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection

Published:2021-06-29 Issue:3 Volume:12 Page:
ISSN:2150-7511
Container-title:mBio
language:en
Short-container-title:mBio

Author:

Sumitomo Tomoko¹^ORCID,Nakata Masanobu¹²^ORCID,Nagase Satoshi³,Takahara Yuki¹,Honda-Ogawa Mariko¹,Mori Yasushi¹,Akamatsu Yukako¹,Yamaguchi Masaya¹^ORCID,Okamoto Shigefumi³⁴^ORCID,Kawabata Shigetada¹

Affiliation:

1. Department of Oral and Molecular Microbiology, Osaka University Graduate School of Dentistry, Osaka, Japan

2. Department of Oral Microbiology, Kagoshima University Graduate School of Medical and Dental Sciences, Kagoshima, Japan

3. Department of Clinical Laboratory Science, Faculty of Health Sciences, Institute of Medical Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan

4. Advanced Health Care Science Research Unit, Innovative Integrated Bio-Research Core, Kanazawa University Institute for Frontier Science Initiative, Kanazawa, Japan

Abstract

Secondary bacterial pneumonia following an influenza A virus (IAV) infection is a major cause of morbidity and mortality. Although it is generally accepted that preceding IAV infection leads to increased susceptibility to secondary bacterial infection, details regarding the pathogenic mechanism during the early stage of superinfection remain elusive.

Funder

Japan Agency for Medical Research and Development

Japan Society for the Promotion of Science

MEXT | Japan Society for the Promotion of Science

GlaxoSmithKline Japan

Takeda Science Foundation

Kobayashi International Scholarship Foundation

Naito Foundation

Publisher

American Society for Microbiology

Subject

Virology,Microbiology