Echinostoma caproni(Trematoda): differentialin vivomucin expression and glycosylation in high- and low-compatible hosts-Reference-Cited by-同舟云学术

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Echinostoma caproni(Trematoda): differentialin vivomucin expression and glycosylation in high- and low-compatible hosts

Published:2014-12-29 Issue:1 Volume:37 Page:32-42
ISSN:0141-9838
Container-title:Parasite Immunology
language:en
Short-container-title:Parasite Immunol

Author:

Cortés A.¹,Muñoz-Antoli C.¹,Sotillo J.¹²,Fried B.³,Esteban J. G.¹,Toledo R.¹

Affiliation:

1. Departamento de Parasitología; Facultad de Farmacia; Universidad de Valencia; Burjassot Valencia Spain

2. Centre for Biodiscovery and Molecular Development of Therapeutics; James Cook University; Smithfield Qld Australia

3. Department of Biology; Lafayette College; Easton PA USA

Funder

Ministerio de EconomÃa y Competitividad

Universidad de Valencia

Publisher

Wiley

Subject

Immunology,Parasitology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/pim.12159/fullpdf

Reference48 articles.

1. Mucin dynamics and enteric pathogens;McGuckin;Nat Rev Microbiol,2011

2. Intestinal nematode parasites, cytokines and effector mechanisms;Else;Int J Parasitol,1998

3. Stat6 dependent goblet cell hyperplasia during intestinal nematode infection;Khan;Parasite Immunol,2001

4. Expulsion of Echinostoma trivolvis (cort, 1914) Kanev, 1985 and retention of E. caproni Richard, 1964 (trematoda: Echinostomatidae) in C3H mice: pathological, ultrastructural, and cytochemical effects on the host intestine;Fujino;Parasitol Res,1993

5. Rapid expulsion of the intestinal trematodes Echinostoma trivolvis and E. caproni from C3H mice by trapping with increased goblet cell mucins;Fujino;Int J Parasitol,1996

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