In Human Immunodeficiency Virus primary infection, early combined antiretroviral therapy reduced γδ T‐cell activation but failed to restore their polyfunctionality
Author:
Affiliation:
1. Laboratory of Cellular Immunology and Pharmacology National Institute for Infectious Diseases “Lazzaro Spallanzani” – IRCCS RomeItaly
2. Clinical Department National Institute for Infectious Diseases “Lazzaro Spallanzani” Rome Italy
Funder
Ministero della Salute
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/imm.13089
Reference25 articles.
1. The multifunctionality of human Vγ9Vδ2 γδ T cells: clonal plasticity or distinct subsets?;Kabelitz D;Scand J Immunol,2012
2. γδ T cell effector functions: a blend of innate programming and acquired plasticity;Bonneville M;Nat Rev Immunol,2010
3. Key features of γδ T‐cell subsets in human diseases and their immunotherapeutic implications;Lawand M;Front Immunol,2017
4. Antiviral reactivities of γδ T cells;Poccia F;Microbes Infect,2005
5. Innate T‐cell immunity in HIV infection: the role of Vγ9Vδ2 T lymphocytes;Poccia F;Curr Mol Med,2002
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