Input-specific plasticity ofN-methyl-d-aspartate receptor-mediated synaptic responses in neonatal rat motoneurons
Author:
Publisher
Wiley
Subject
General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1460-9568.2009.06769.x/fullpdf
Reference46 articles.
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2. Acute modulation of synaptic transmission to motoneurons by BDNF in the neonatal rat spinal cord;Arvanian;Eur. J. Neurosci.,2001
3. Removal of NMDA receptor Mg2+ block extends the action of neurotrophin-3 on synaptic transmission in neonatal rat motoneurons;Arvanian;J. Neurophysiol.,2001
4. Viral delivery of NR2D subunits reduces Mg2+ block of NMDA receptor and restores NT-3-induced potentiation of AMPA/kainate responses in maturing rat motoneurons;Arvanian;J. Neurophysiol.,2004
5. Comparison of metabotropic glutamate receptor responses at segmental and descending inputs to motoneurons in neonatal rat spinal cord;Arvanian;J. Pharmacol. Exp. Ther.,2005
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2. Involvement of N-methyl-d-aspartate receptors in plasticity induced by paired corticospinal-motoneuronal stimulation in humans;Journal of Neurophysiology;2018-02-01
3. Neurotrophic Factors in Spinal Cord Injury;Neurotrophic Factors;2014
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