N-methyl-d-aspartate receptor- and metabotropic glutamate receptor-dependent long-term depression are differentially regulated by the ubiquitin-proteasome system-Reference-Cited by-同舟云学术

N-methyl-d-aspartate receptor- and metabotropic glutamate receptor-dependent long-term depression are differentially regulated by the ubiquitin-proteasome system

Published:2009-10 Issue:8 Volume:30 Page:1443-1450
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:

Author:

Citri Ami,Soler-Llavina Gilberto,Bhattacharyya Samarjit,Malenka Robert C.

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1460-9568.2009.06950.x/fullpdf

Reference44 articles.

1. Regulation of the level of Vesl-1S/Homer-1a proteins by ubiquitin-proteasome proteolytic systems;Ageta;J. Biol. Chem.,2001

2. Metabotropic glutamate receptors contribute to the induction of long-term depression in the CA1 region of the hippocampus;Bashir;Eur. J. Pharmacol.,1993

3. The mGluR theory of fragile X mental retardation;Bear;Trends Neurosci.,2004

4. Regulation of AMPA receptor endocytosis by a signaling mechanism shared with LTD;Beattie;Nat. Neurosci.,2000

5. Mechanisms of synaptic depression triggered by metabotropic glutamate receptors;Bellone;Cell. Mol. Life Sci.,2008

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