Historical perspective and progress on protein ubiquitination at glutamatergic synapses-Reference-Cited by-同舟云学术

Historical perspective and progress on protein ubiquitination at glutamatergic synapses

Published:2021-09 Issue: Volume:196 Page:108690
ISSN:0028-3908
Container-title:Neuropharmacology
language:en
Short-container-title:Neuropharmacology

Author:

Mabb Angela M.^ORCID

Funder

National Institute of Neurological Disorders and Stroke

National Science Foundation Directorate for Biological Sciences

National Science Foundation

Brain and Behavior Research Foundation

Whitehall Foundation

National Alliance for Research on Schizophrenia and Depression

National Institutes of Health

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Pharmacology

Reference259 articles.

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3. Synaptic Drosophila UNC-13 is regulated by antagonistic G-protein pathways via a proteasome-dependent degradation mechanism;Aravamudan;J. Neurobiol.,2003

4. Loss of F-box only protein 2 (Fbxo2) disrupts levels and localization of select NMDA receptor subunits, and promotes aberrant synaptic connectivity;Atkin;J. Neurosci.,2015

5. The Phr1 ubiquitin ligase promotes injury-induced axon self-destruction;Babetto;Cell Rep.,2013

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