Transfer of rapid inactivation and sensitivity to the class III antiarrhythmic drug E-4031 from HERG to M-eag channels
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1469-7793.1998.003bi.x/fullpdf
Reference47 articles.
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2. Chimeric dopamine-norepinephrine transporters delineate structural domains influencing selectivity for catecholamines and 1-methyl-4-phenylpyridinium;Buck;Proceedings of the National Academy of Sciences of the USA,1994
3. Tetraethylammonium blockade distinguishes two inactivation mechanisms in voltage-activated K channels;Choi;Proceedings of the National Academy of Sciences of the USA,1991
4. A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome;Curran;Cell,1995
5. Inactivation determined by a single site in K pores;Biasi;Pflügers Archiv,1993
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