PML2‐mediated thread‐like nuclear bodies mark late senescence in Hutchinson–Gilford progeria syndrome-Reference-Cited by-同舟云学术

PML2‐mediated thread‐like nuclear bodies mark late senescence in Hutchinson–Gilford progeria syndrome

Published:2020-04-29 Issue:6 Volume:19 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Wang Ming¹²³,Wang Lulu³,Qian Minxian¹²^ORCID,Tang Xiaolong¹²,Liu Zuojun¹²,Lai Yiwei³,Ao Ying¹²,Huang Yinghua³,Meng Yuan¹²,Shi Lei¹²,Peng Linyuan¹²,Cao Xinyue¹²,Wang Zimei¹²⁴,Qin Baoming³,Liu Baohua¹²⁴⁵^ORCID

Affiliation:

1. Shenzhen Key Laboratory for Systemic Aging and Intervention (SAI) National Engineering Research Center for Biotechnology (Shenzhen) Medical Research Center Shenzhen University Health Science Center Shenzhen China

2. Guangdong Key Laboratory for Genome Stability and Human Disease Prevention Department of Biochemistry & Molecular Biology School of Basic Medical Sciences Shenzhen University Health Science Center Shenzhen China

3. Guangdong Provincial Key Laboratory of Stem Cell and Regenerative Medicine Guangzhou Institutes of Biomedicine and Health Chinese Academy of Sciences Guangzhou China

4. Carson International Cancer Center Shenzhen University Health Science Center Shenzhen China

5. Guangdong Provincial Key Laboratory of Regional Immunity and Diseases School of Basic Medical Sciences Shenzhen University Health Science Center Shenzhen China

Funder

National Natural Science Foundation of China

China Postdoctoral Science Foundation

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13147

Reference38 articles.

1. Lamin A buffers CK2 kinase activity to modulate aging in a progeria mouse model.

2. Nuclear and chromatin reorganization in the MHC-Oct3/4 locus at developmental phases of embryonic stem cell differentiation