Progress and trends in the development of therapies for Hutchinson–Gilford progeria syndrome
Author:
Affiliation:
1. School of Life and Health Sciences The Chinese University of Hong Kong (Shenzhen) Shenzhen China
2. Department of Applied Biology and Chemical Technology Hong Kong Polytechnic University Hong Kong Special Administrative Region China
Funder
Natural Science Foundation of Guangdong Province
Publisher
Wiley
Subject
Cell Biology,Aging
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13175
Reference93 articles.
1. Inhibition of DNA damage response at telomeres improves the detrimental phenotypes of Hutchinson–Gilford Progeria Syndrome
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4. Lamin A buffers CK2 kinase activity to modulate aging in a progeria mouse model.
5. Rapamycin: An InhibiTOR of Aging Emerges From the Soil of Easter Island
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