Influenza A virus NS1 protein-induced JNK activation and apoptosis are not functionally linked
Author:
Affiliation:
1. Institute of Virology (IVM), University Hospital Münster; WWU; Germany
2. Cluster of Excellence “Cells in Motion”; University of Muenster; Germany
3. Interdisciplinary Center of Clinical Research (IZKF), UKM; WWU; Germany
Funder
Deutsche Forschungsgemeinschaft
Publisher
Hindawi Limited
Subject
Virology,Immunology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/cmi.12721/fullpdf
Reference39 articles.
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2. Upregulation of cleavage and polyadenylation specific factor 4 in lung adenocarcinoma and its critical role for cancer cell survival and proliferation;Chen;PloS One,2013
3. Influenza A/Hong Kong/156/1997(H5N1) virus NS1 gene mutations F103L and M106I both increase IFN antagonism, virulence and cytoplasmic localization but differ in binding to RIG-I and CPSF30;Dankar;Virology Journal,2013
4. Influenza A virus NS1 protein activates the PI3K/Akt pathway to mediate antiapoptotic signaling responses;Ehrhardt;Journal of Virology,2007
5. Ligand-activated site-specific recombination in mice;Feil;Proceedings of the National Academy of Sciences of the United States of America,1996
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