The gap junction modifier ZP1609 decreases cardiomyocyte hypercontracture following ischaemia/reperfusion independent from mitochondrial connexin 43
Author:
Affiliation:
1. Physiologisches Institut; Justus-Liebig-Universität Giessen; Giessen Germany
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.13804/fullpdf
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5. Identification of ischemia-regulated phosphorylation sites in connexin43: a possible target for the antiarrhythmic peptide analogue rotigaptide (ZP123);Axelsen;J Mol Cell Cardiol,2006
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