Platelet activation by dimeric2-glycoproteinI requires signaling via both glycoproteinIb and apolipoproteinE receptor2
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1538-7836.2008.03021.x/fullpdf
Reference34 articles.
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2. Pathophysiology of the antiphospholipid syndrome;de Groot;J Thromb Haemost,2005
3. The binding site in beta2-glycoproteinI for ApoER2 on platelets is located in domainV;van Lummel;J Biol Chem,2005
4. Dimers of beta2-glycoproteinI increase platelet deposition to collagen via interaction with phospholipids and the apolipoproteinE receptor2;Lutters;J Biol Chem,2003
5. Platelet adhesion to dimeric beta2-glycoprotein-I under conditions of flow is mediated by at least two receptors: glycoprotein Ibalpha and APOER2;Pennings;J Thromb Haemost,2007
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