Inactivation of ADAMTS13 by plasmin as a potential cause of thrombotic thrombocytopenic purpura
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1538-7836.2010.03942.x/fullpdf
Reference46 articles.
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2. Mutations in a member of the ADAMTS gene family cause thrombotic thrombocytopenic purpura;Levy;Nature,2001
3. Effect of plasma exchange on plasma ADAMTS13 metalloprotease activity, inhibitor level, and clinical outcome in patients with idiopathic and nonidiopathic thrombotic thrombocytopenic purpura;Zheng;Blood,2004
4. von Willebrand factor cleaving protease (ADAMTS-13) and ADAMTS-13 neutralizing autoantibodies in 100 patients with thrombotic thrombocytopenic purpura;Peyvandi;Br J Haematol,2004
5. Specific von Willebrand factor-cleaving protease in thrombotic microangiopathies: a study of 111 cases;Veyradier;Blood,2001
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1. Optimization of plasma-based BioID identifies plasminogen as a ligand of ADAMTS13;Scientific Reports;2024-04-20
2. Hemophagocytic lymphohistiocytosis is associated with deficiency and closed conformation of ADAMTS-13;Research and Practice in Thrombosis and Haemostasis;2024-01
3. Mechanisms of ADAMTS13 regulation;Journal of Thrombosis and Haemostasis;2022-12
4. Low ADAMTS13 Activity Correlates with Increased Mortality in COVID-19 Patients;TH Open;2021-01
5. Evidence for secondary thrombotic microangiopathy in COVID-19;2020-10-23
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