EGCG inhibits pressure overload‐induced cardiac hypertrophy via the PSMB5/Nmnat2/SIRT6‐dependent signalling pathways-Reference-Cited by-同舟云学术

EGCG inhibits pressure overload‐induced cardiac hypertrophy via the PSMB5/Nmnat2/SIRT6‐dependent signalling pathways

Published:2021-01-07 Issue:4 Volume:231 Page:
ISSN:1748-1708
Container-title:Acta Physiologica
language:en
Short-container-title:Acta Physiol.

Author:

Cai Yi¹²³^ORCID,Yu Shan Shan²⁴,He Yang⁵⁶,Bi Xue Ying²,Gao Si²,Yan Ting Dong⁷,Zheng Guo Dong¹,Chen Ting Ting²,Ye Jian Tao²,Liu Pei Qing²

Affiliation:

1. Key Laboratory of Molecular Target & Clinical Pharmacology and the State Key Laboratory of Respiratory Disease School of Pharmaceutical Sciences & the Fifth Affiliated Hospital Guangzhou Medical University Guangzhou China

2. Department of Pharmacology and Toxicology School of Pharmaceutical Sciences Sun Yat‐Sen University Guangzhou China

3. Cancer and Stem Cell Biology Program Duke‐NUS Medical School Singapore Singapore

4. Laboratory of Research of New Chinese Medicine Department of Pharmacy Zhujiang HospitalSouthern Medical University Guangzhou China

5. BayRay Innovation CenterShenzhen Bay Laboratory Shenzhen China

6. Institute of Molecular and Cell Biology Singapore Singapore

7. Department of Pharmacology School of Pharmacy Nantong University Nantong China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/apha.13602

Reference42 articles.

1. Metabolic Remodeling Promotes Cardiac Hypertrophy by Directing Glucose to Aspartate Biosynthesis

2. Cardiomyocyte Sirt (Sirtuin) 7 Ameliorates Stress-Induced Cardiac Hypertrophy by Interacting With and Deacetylating GATA4

3. Lansoprazole alleviates pressure overload-induced cardiac hypertrophy and heart failure in mice by blocking the activation of β-catenin

4. Nicotinamide adenine dinucleotide induces a bivalent metabolism and maintains pluripotency in human embryonic stem cells

5. Metabolism and biochemical properties of nicotinamide adenine dinucleotide (NAD) analogs, nicotinamide guanine dinucleotide (NGD) and nicotinamide hypoxanthine dinucleotide (NHD)

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