CCL7 contributes to angiotensin II‐ induced abdominal aortic aneurysm by promoting macrophage infiltration and pro‐inflammatory phenotype-Reference-Cited by-同舟云学术

CCL7 contributes to angiotensin II‐ induced abdominal aortic aneurysm by promoting macrophage infiltration and pro‐inflammatory phenotype

Published:2021-06-29 Issue:15 Volume:25 Page:7280-7293
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Xie Cuiping¹,Ye Feiming¹,Zhang Ning¹,Huang Yuxue¹,Pan Yun²,Xie Xiaojie¹^ORCID

Affiliation:

1. Department of Cardiology Cardiovascular Key Laboratory of Zhejiang Province Zhejiang University School of Medicine Second Affiliated Hospital Hangzhou China

2. College of Information Science and Electronic Engineering Zhejiang University Hangzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.16757

Reference51 articles.

1. Abdominal aortic aneurysm

2. Pathophysiology and epidemiology of abdominal aortic aneurysms

3. Monocytes and macrophages in abdominal aortic aneurysm

4. Macrophage Polarization

5. B lymphocytes trigger monocyte mobilization and impair heart function after acute myocardial infarction

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