Angiotensin receptor–neprilysin inhibitor (thiorphan/irbesartan) improved cardiac function in a rat model of myocardial ischemic reperfusion injury-Reference-Cited by-同舟云学术

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Angiotensin receptor–neprilysin inhibitor (thiorphan/irbesartan) improved cardiac function in a rat model of myocardial ischemic reperfusion injury

Published:2022-08-02 Issue:1 Volume:37 Page:31-43
ISSN:0767-3981
Container-title:Fundamental & Clinical Pharmacology
language:en
Short-container-title:Fundamemntal Clinical Pharma

Author:

Mohammed Abdulsalam Takwa¹,Hasanin Amany H.¹,Hussein Mohamed Reham¹^ORCID,Khairy Eman²,Mahmoud Dalia²,Habib Eman³,Badawy Ahmed El Sayed¹

Affiliation:

1. Department of Clinical Pharmacology, Faculty of Medicine Ain Shams University Cairo Egypt

2. Department of Medical Biochemistry and Molecular Biology, Faculty of Medicine Ain Shams University Cairo Egypt

3. Department of Anatomy and Embryology, Faculty of Medicine Ain Shams University Cairo Egypt

Publisher

Wiley

Subject

Pharmacology (medical),Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/fcp.12818

Reference47 articles.

1. Ischemic Heart Disease: An Update

2. Vitexin attenuates myocardial ischemia/reperfusion injury in rats by regulating mitochondrial dysfunction induced by mitochondrial dynamics imbalance

3. ROS and redox signaling in myocardial ischemia-reperfusion injury and cardioprotection

4. Cardioprotective signalling: Past, present and future

5. Mff‐Dependent Mitochondrial Fission Contributes to the Pathogenesis of Cardiac Microvasculature Ischemia/Reperfusion Injury via Induction of mROS‐Mediated Cardiolipin Oxidation and HK2/VDAC1 Disassociation‐Involved mPTP Opening

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. A comparative study between angiotensin receptor neprilysin inhibitor (thiorphan/irbesartan) with each of nitrate and carvedilol in a rat model of myocardial ischemic reperfusion injury;Fundamental & Clinical Pharmacology;2023-03-15

2. Salidroside Inhibits Ischemia/Reperfusion-Induced Myocardial Apoptosis by Targeting Mir-378a-3p Via the Igf1r/Pi3k/Akt Signaling Pathway;Transplantation Proceedings;2022-09

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