Aryl hydrocarbon receptor activation by diesel exhaust particles mediates epithelium-derived cytokines expression in severe allergic asthma

Author:

Weng C.-M.1,Wang C.-H.23,Lee M.-J.1,He J.-R.3,Huang H.-Y.3,Chao M.-W.4,Chung K. F.56,Kuo H.-P.1ORCID

Affiliation:

1. Department of Medicine; Taipei Medical University College of Medicine; Taipei Taiwan

2. Department of Medicine; Chang Gung University College of Medicine; Taoyuan Taiwan

3. Department of Thoracic Medicine; Chang Gung Memorial Hospital; Chang Gung Medical Foundation; Taoyuan Taiwan

4. Center of Nanotechnology; Chung Yuan Christian University; Zhongli District, Taoyuan City Taiwan

5. Airway Disease Section; National Heart and Lung Institute; Imperial College London; London UK

6. NIHR Respiratory Biomedical Research Unit; Royal Brompton NHS Foundation Trust; London UK

Funder

Taiwan Ministry of Science and Technology

Chang Gung Medical Foundation

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference30 articles.

1. International ERS/ATS guidelines on definition, evaluation and treatment of severe asthma;Chung;Eur Respir J,2014

2. Measures of gene expression in sputum cells can identify TH2-high and TH2-low subtypes of asthma;Peters;J Allergy Clin Immunol,2014

3. Dynamic role of epithelium-derived cytokines in asthma;Bartemes;Clin Immunol,2012

4. Interplay of air pollution and asthma immunopathogenesis: a focused review of diesel exhaust and ozone;Alexis;Int Immunopharmacol,2014

5. Link between environmental air pollution and allergic asthma: east meets West;Zhang;J Thorac Dis,2015

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