Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndrome-Reference-Cited by-同舟云学术

Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndrome

Published:2019-07-31 Issue:10 Volume:23 Page:6543-6553
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Larionov Alexey¹,Dahlke Eileen²,Kunke Madlen²,Zanon Rodriguez Luis²,Schiessl Ina M.³,Magnin Jean‐Luc⁴,Kern Ursula⁵,Alli Abdel A.⁶,Mollet Geraldine⁷,Schilling Oliver⁵⁸,Castrop Hayo³,Theilig Franziska¹²^ORCID

Affiliation:

1. Institute of Anatomy, Department of Medicine University of Fribourg Fribourg Switzerland

2. Institute of Anatomy Christian Albrechts‐University Kiel Kiel Germany

3. Institute of Physiology University of Regensburg Regensburg Germany

4. Service LaboratoireHôpital Fribourg Fribourg Switzerland

5. Institute of Molecular Medicine and Cell Research, Faculty of Medicine University of Freiburg Freiburg Germany

6. Department of Physiology and Functional Genomics University of Florida Gainesville Florida

7. Laboratory of Hereditary Kidney Diseases, INSERM UMR 1163 Université Paris Descartes‐Sorbonne Paris Cité, Imagine Institute Paris France

8. BIOSS Center for Biological Signaling Studies University of Freiburg Freiburg Germany

Funder

Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.14387

Reference27 articles.

1. Clinical and epidemiological assessment of steroid-resistant nephrotic syndrome associated with the NPHS2 R229Q variant

2. NPHS2Variation in Sporadic Focal Segmental Glomerulosclerosis

3. Podocin Inactivation in Mature Kidneys Causes Focal Segmental Glomerulosclerosis and Nephrotic Syndrome

4. Role for intrarenal mechanisms in the impaired salt excretion of experimental nephrotic syndrome.

5. Collecting duct is a site of sodium retention in PAN nephrosis: a rationale for amiloride therapy;Deschenes G;J Am Soc Nephrol,2001

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