The mechanism ofATF3repression of epithelial-mesenchymal transition and suppression of cell viability in cholangiocarcinoma viap53signal pathway
Author:
Affiliation:
1. Department of Biliary Surgery; West China Hospital of Sichuan University; Chengdu Sichuan China
2. Ambulatory Surgery Center; West China Hospital of Sichuan University; Chengdu Sichuan China
Funder
Science and Technology Office of Sichuan Province
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/jcmm.14132/fullpdf
Reference25 articles.
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2. Effect of Mir-122 on human cholangiocarcinoma proliferation, invasion, and apoptosis through P53 expression;Wu;Med Sci Monit,2016
3. aPKC-iota/P-Sp1/Snail signaling induces epithelial-mesenchymal transition and immunosuppression in cholangiocarcinoma;Qian;Hepatology,2017
4. Abnormal expression of Forkhead Box J2 (FOXJ2) suppresses migration and invasion in extrahepatic cholangiocarcinoma and is associated with prognosis;Qiang;Int J Oncol,2015
5. miR-21 and KLF4 jointly augment epithelialmesenchymal transition via the Akt/ERK1/2 pathway;Liu;Int J Oncol,2017
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