JS‐K induces reactive oxygen species‐dependent anti‐cancer effects by targeting mitochondria respiratory chain complexes in gastric cancer
Author:
Affiliation:
1. Department of General Surgery Chinese People's Liberation Army General Hospital Beijing China
2. Institute of Military Cognitive and Brain Sciences Academy of Military Medical Sciences Beijing China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.14122
Reference43 articles.
1. The Nitric Oxide Prodrug JS-K and Its Structural Analogues as Cancer Therapeutic Agents
2. Antitumor Activity of JS-K [O2-(2,4-Dinitrophenyl) 1-[(4-Ethoxycarbonyl)piperazin-1-yl]diazen-1-ium-1,2-diolate] and Related O2-Aryl Diazeniumdiolates in Vitro and in Vivo
3. JS‐K, a glutathione/glutathione S‐transferase‐activated nitric oxide donor of the diazeniumdiolate class with potent antineoplastic activity;Shami PJ;Mol Cancer Ther,2003
4. JS-K, a nitric oxide prodrug, induces cytochrome c release and caspase activation in HL-60 myeloid leukemia cells
5. JS-K promotes apoptosis by inducing ROS production in human prostate cancer cells
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