Mitochondria, lysosomes, and dysfunction: their meaning in neurodegeneration-Reference-Cited by-同舟云学术

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Mitochondria, lysosomes, and dysfunction: their meaning in neurodegeneration

Published:2018-08-02 Issue:3 Volume:147 Page:291-309
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Audano Matteo¹,Schneider Anja²³,Mitro Nico¹^ORCID

Affiliation:

1. DiSFeB; Dipartimento di Scienze Farmacologiche e Biomolecolari; Università degli Studi di Milano; Milano Italy

2. German Center for Neurodegenerative Diseases; DZNE; Bonn Germany

3. Department for Neurodegenerative Diseases and Geriatric Psychiatry; University Clinic; Bonn Germany

Funder

Fondazione Cariplo

European Foundation for the Study of Diabetes

Ministero dell’Istruzione, dell’Università e della Ricerca

Deutsche Forschungsgemeinschaft

Michael J. Fox Foundation for Parkinson's Research

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.14471/fullpdf

Reference162 articles.

1. Lysosomal metabolomics reveals V-ATPase-and mTOR-dependent regulation of amino acid efflux from lysosomes;Abu-Remaileh;Science,2017

2. Decreased proteolytic activity of the mitochondrial amyloid-beta degrading enzyme, PreP peptidasome, in Alzheimer's disease brain mitochondria;Alikhani;J. Alzheimer's Dis.,2011

3. A mutation in sigma-1 receptor causes juvenile amyotrophic lateral sclerosis;Al-Saif;Ann. Neurol.,2011

4. Role of the mammalian retromer in sorting of the cation-independent mannose 6-phosphate receptor;Arighi;J. Cell. Biol.,2004

5. Lysosomal storage disease: gene therapy on both sides of the blood-brain barrier;Aronovich;Mol. Genet. Metab.,2015

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