Further evidence for a differential interaction of brivaracetam and levetiracetam with the synaptic vesicle 2A protein
Author:
Affiliation:
1. UCB BioPharma SPRL; Braine-l'Alleud Belgium
Funder
UCB Pharma
Publisher
Wiley
Subject
Clinical Neurology,Neurology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/epi.14532/fullpdf
Reference12 articles.
1. Brivaracetam: rationale for discovery and preclinical profile of a selective SV2A ligand for epilepsy treatment;Klitgaard;Epilepsia,2016
2. Modulation of the conformational state of the SV2A protein by an allosteric mechanism as evidenced by ligand binding assays;Daniels;Br J Pharmacol,2013
3. Evidence for a differential interaction of brivaracetam and levetiracetam with the synaptic vesicle 2A protein;Wood;Epilepsia,2017
4. SV2C is a synaptic vesicle protein with an unusually restricted localization: anatomy of a synaptic vesicle protein family;Janz;Neuroscience,1999
5. Synaptic vesicle protein 2A: basic facts and role in synaptic function;Mendoza-Torreblanca;Eur J Neurosci,2013
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2. Structures of synaptic vesicle protein 2A and 2B bound to anticonvulsants;Nature Structural & Molecular Biology;2024-06-19
3. Structural basis for antiepileptic drugs and botulinum neurotoxin recognition of SV2A;Nature Communications;2024-04-18
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