<scp> APOE </scp> ɛ4 allele and <scp> TOMM40 ‐ APOC1 </scp> variants jointly contribute to survival to older ages-Reference-Cited by-同舟云学术

APOE ɛ4 allele and TOMM40 ‐ APOC1 variants jointly contribute to survival to older ages

Published:2022-11-03 Issue:12 Volume:21 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Kulminski Alexander M.¹,Jain‐Washburn Ethan¹,Philipp Ian¹,He Liang¹,Loika Yury¹,Loiko Elena¹,Bagley Olivia¹,Ukraintseva Svetlana¹,Yashin Anatoliy¹,Arbeev Konstantin¹,Stallard Eric¹,Feitosa Mary F.²,Schupf Nicole³,Christensen Kaare⁴,Culminskaya Irina¹

Affiliation:

1. Biodemography of Aging Research Unit, Social Science Research Institute Duke University Durham North Carolina USA

2. Division of Statistical Genomics, Department of Genetics Washington University School of Medicine St Louis Missouri USA

3. Gertrude H. Sergievsky Center Columbia University Irving Medical Center New York New York USA

4. Unit of Epidemiology, Biostatistics and Biodemography, Department of Public Health Southern Denmark University Odense Denmark

Funder

National Institute on Aging

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13730

Reference69 articles.

1. Effect of APOE ε4 genotype on amyloid-β and tau accumulation in Alzheimer’s disease

2. Network biology: understanding the cell's functional organization

3. Functional analysis of APOE locus genetic variation implicates regional enhancers in the regulation of both TOMM40 and APOE

4. A Quarter Century of APOE and Alzheimer’s Disease: Progress to Date and the Path Forward

5. Lipid- and receptor-binding regions of apolipoprotein E4 fragments act in concert to cause mitochondrial dysfunction and neurotoxicity

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