Autophagy promotes phagocytosis and clearance of Treponema pallidum via the NLRP3 inflammasome in macrophages
Author:
Affiliation:
1. Center of Clinical Laboratory Zhongshan Hospital School of Medicine Xiamen University Xiamen China
2. Institute of Infectious Disease School of Medicine Xiamen University Xiamen China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Infectious Diseases,Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jdv.16463
Reference22 articles.
1. Profile of the tprK gene in primary syphilis patients based on next-generation sequencing
2. Development of tissue inflammation accompanied by NLRP3 inflammasome activation in rabbits infected with Treponema pallidum strain Nichols
3. Akt, mTOR and NF-κB pathway activation in Treponema pallidum stimulates M1 macrophages
4. Phagocytosis of Borrelia burgdorferi and Treponema pallidum Potentiates Innate Immune Activation and Induces Gamma Interferon Production
5. Deletion ofNlrp3Augments Survival during Polymicrobial Sepsis by Decreasing Autophagy and Enhancing Phagocytosis
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1. Endothelial dysfunction of syphilis: Pathogenesis;Journal of the European Academy of Dermatology and Venereology;2024-02-20
2. Proinflammatory CD14 high CD16 low monocytes/macrophages prevail in Treponema phagedenis- associated bovine digital dermatitis;Infection and Immunity;2024-02-13
3. Treponema pallidum recombinant protein Tp47 activates NOD-like receptor family protein 3 inflammasomes in macrophages via glycolysis;International Immunopharmacology;2024-01
4. Treponema pallidum promoted microglia apoptosis and prevented itself from clearing by human microglia via blocking autophagic flux;PLOS Pathogens;2023-08-23
5. Recombinant Treponema pallidum protein Tp47 promoted the phagocytosis of macrophages by activating NLRP3 inflammasome induced by PKM2‐dependent glycolysis;Journal of the European Academy of Dermatology and Venereology;2023-06-12
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