Cooperation of Sall4 and Sox8 transcription factors in the regulation of the chicken Sox3 gene during otic placode development
Author:
Affiliation:
1. Graduate School of Frontier Biosciences; Osaka University; Osaka Japan
2. School of Environmental Science and Engineering; Kochi University of Technology; Kochi Japan
3. Faculty of Life Sciences; Kyoto Sangyo University; Kyoto Japan
Funder
Japan Society for the Promotion of Science
Ministry of Education, Culture, Sports, Science and Technology
Publisher
Wiley
Subject
Cell Biology,Developmental Biology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/dgd.12427/fullpdf
Reference47 articles.
1. Eya1-Six1 interaction is sufficient to induce hair cell fate in the cochlea by activating Atoh1 expression in cooperation with Sox2;Ahmed;Developmental Cell,2012
2. Spalt4 mediates invagination and otic placode gene expression in cranial ectoderm;Barembaum;Development,2007
3. A Sox10 enhancer element common to the otic placode and neural crest is activated by tissue-specific paralogs;Betancur;Development,2011
4. SALL4 deletions are a common cause of Okihiro and acro-renal-ocular syndromes and confirm haploinsufficiency as the pathogenic mechanism;Borozdin;Journal of Medical Genetics,2004
5. Adjacent DNA sequences modulate Sox9 transcriptional activation at paired Sox sites in three chondrocyte-specific enhancer elements;Bridgewater;Nucleic Acids Research,2003
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