Substantially elevating the levels of αB-crystallin in spinal motor neurons of mutant SOD1 mice does not significantly delay paralysis or attenuate mutant protein aggregation
Author:
Affiliation:
1. Department of Neuroscience; Center for Translational Research in Neurodegenerative Disease; McKnight Brain Institute; University of Florida; Gainesville Florida USA
Funder
National Institutes of Neurological Disease and Stroke
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13022/fullpdf
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