Ca v 3.2 T‐type calcium channel mutation influences kindling‐induced thalamic neuronal firing patterns in Genetic Absence Epilepsy Rats From Strasbourg-Reference-Cited by-同舟云学术

Ca v 3.2 T‐type calcium channel mutation influences kindling‐induced thalamic neuronal firing patterns in Genetic Absence Epilepsy Rats From Strasbourg

Published:2019-06-17 Issue:7 Volume:60 Page:1378-1386
ISSN:0013-9580
Container-title:Epilepsia
language:en
Short-container-title:Epilepsia

Author:

Çarçak Nihan¹²^ORCID,Ali Idrish²³,Powell Kim²³,Zheng Thomas²,Onat Filiz⁴⁵,O'Brien Terence J.²³

Affiliation:

1. Department of Pharmacology Faculty of Pharmacy Istanbul University Istanbul Turkey

2. Departments of Medicine and Neurology Royal Melbourne Hospital University of Melbourne Melbourne Victoria Australia

3. Departments of Neuroscience and Neurology Central Clinical School Alfred Health Monash University Clayton Victoria Australia

4. Department of Pharmacology and Clinical Pharmacology Faculty of Medicine Marmara University Istanbul Turkey

5. Marmara University Epilepsy Research Center Istanbul Turkey

Funder

National Health and Medical Research Council

Publisher

Wiley

Subject

Clinical Neurology,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/epi.16076

Reference32 articles.

1. The effect of amygdala kindling on neuronal firing patterns in the lateral thalamus in the GAERS model of absence epilepsy

2. Resistance to Propagation of Amygdaloid Kindling Seizures in Rats with Genetic Absence Epilepsy

3. Suppression of limbic motor seizures by electrical stimulation in thalamic reticular nucleus

4. Selective increase in T-type calcium conductance of reticular thalamic neurons in a rat model of absence epilepsy

5. Low-voltage-activated calcium channel subunit expression in a genetic model of absence epilepsy in the rat

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