Pancreatitis severity in mice with impaired CFTR function but pancreatic sufficiency is mediated via ductal and inflammatory cells‐Not acinar cells-Reference-Cited by-同舟云学术

Pancreatitis severity in mice with impaired CFTR function but pancreatic sufficiency is mediated via ductal and inflammatory cells‐Not acinar cells

Published:2021-03-08 Issue: Volume: Page:
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Trapp Simon¹,Aghdassi Ali A.¹^ORCID,Glaubitz Juliane¹,Sendler Matthias¹,Weiss Frank Ulrich¹,Kühn Jens Peter²,Kromrey Marie‐Luise²,Mahajan Ujjwal M.¹³,Pallagi Petra⁴,Rakonczay Zoltán⁵,Venglovecz Viktória⁶,Lerch Markus M.¹,Hegyi Peter⁷,Mayerle Julia¹³

Affiliation:

1. Department of Medicine A University Medicine Greifswald Greifswald Germany

2. Institute of Diagnostic Radiology and Neuroradiology University Medicine Greifswald Greifswald Germany

3. Department of Medicine II Ludwig‐Maximilians University Munich Munich Germany

4. First Department of Medicine University of Szeged Szeged Hungary

5. Department of Pathophysiology University of Szeged Szeged Hungary

6. Department of Pharmacology and Pharmacotherapy University of Szeged Szeged Hungary

7. Department of Translational Medicine/First Department of Medicine Medical School Institute for Translational Medicine Pécs Hungary

Funder

Deutsche Forschungsgemeinschaft

Bundesministerium für Bildung und Forschung

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.16404

Reference58 articles.

1. English language version of the S3-consensus guidelines on chronic pancreatitis: Definition, aetiology, diagnostic examinations, medical, endoscopic and surgical management of chronic pancreatitis

2. Hereditary pancreatitis caused by mutation-induced misfolding of human cationic trypsinogen: A novel disease mechanism

3. CCK-JMV-180, an analog of cholecystokinin, releases intracellular calcium from an inositol trisphosphate-independent pool in rat pancreatic acini.

4. Trypsin activity is not involved in premature, intrapancreatic trypsinogen activation

5. Cathepsin L Inactivates Human Trypsinogen, Whereas Cathepsin L-Deletion Reduces the Severity of Pancreatitis in Mice

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