A mechanism for NaV1.5 downregulation and sodium current decrease in heart failure
Author:
Affiliation:
1. Medical Sciences Department; University of Girona Medical School; Girona Spain
2. Girona Biomedical Research Institute; Salt Spain
3. Biomedical Research Networking Center on Cardiovascular Diseases (CIBERCV); Madrid Spain
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/apha.12901/fullpdf
Reference10 articles.
1. Regulation of the cardiac Na+ channel NaV1.5 by post-translational modifications;Marionneau;J Mol Cell Cardiol,2015
2. Calcium-dependent Nedd4-2 upregulation mediates degradation of the cardiac sodium channel NaV1.5: implications for heart failure;Luo;Acta Physiol (Oxf),2017
3. Cardiac NaV1.5 is modulated by ubiquitin protein ligase E3 component n-recognin UBR3 and 6;Zhao;J Cell Mol Med,2015
4. Proteasome inhibitor (MG132) rescues NaV1.5 protein content and the cardiac sodium current in dystrophin-deficient mdx (5cv) mice;Rougier;Front Physiol,2013
5. Regulation of the human ether-a-go-go-related gene (hERG) channel by Rab4 protein through neural precursor cell-expressed developmentally down-regulated protein 4-2 (Nedd4-2);Cui;J Biol Chem,2013
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