Cardiac Nav 1.5 is modulated by ubiquitin protein ligase E3 component n-recognin UBR3 and 6-Reference-Cited by-同舟云学术

Cardiac Nav 1.5 is modulated by ubiquitin protein ligase E3 component n-recognin UBR3 and 6

Published:2015-06-07 Issue:9 Volume:19 Page:2143-2152
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Zhao Chunxia¹²,Wang Lijie¹²,Ma Xiue¹²,Zhu Weidong¹³⁴,Yao Lei¹²³,Cui Yingyu¹⁴⁵,Liu Yi¹³⁵,Li Jun¹³⁵,Liang Xingqun¹³⁴,Sun Yunfu¹³⁴,Li Li¹⁴⁵,Chen Yi-Han¹²³⁴⁵

Affiliation:

1. Key Laboratory of Arrhythmias of the Ministry of Education of China; East Hospital; Tongji University School of Medicine; Shanghai China

2. Department of Cardiology; Shanghai East Hospital; Tongji University School of Medicine; Shanghai China

3. Research Center for Translational Medicine; Tongji University School of Medicine; Shanghai China

4. Department of Pathology and Pathophysiology; Tongji University School of Medicine; Shanghai China

5. Institute of Medical Genetics; Tongji University; Shanghai China

Funder

National Key Basic Research Program of China

National Natural Science Foundation of China

Major International Joint Research Program Fund of China

General Program of National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jcmm.12588/fullpdf

Reference39 articles.

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2. Biology of cardiac sodium channel Nav1.5 expression;Rook;Cardiovasc Res,2012

3. Regulation of voltage-gated ion channels in excitable cells by the ubiquitin ligases Nedd4 and Nedd4-2;Bongiorno;Channels,2011

4. Protein-protein interactions involving voltage-gated sodium channels: post-translational regulation, intracellular trafficking and functional expression;Shao;Int J Biochem Cell Biol,2009

5. Bepridil up-regulates cardiac Na+ channels as a long-term effect by blunting proteasome signals through inhibition of calmodulin activity;Kang;Brit J Pharmacol,2009

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