Sec61 blockade by mycolactone: A central mechanism in Buruli ulcer disease-Reference-Cited by-同舟云学术

Sec61 blockade by mycolactone: A central mechanism in Buruli ulcer disease

Published:2018-08-14 Issue:11 Volume:110 Page:237-248
ISSN:0248-4900
Container-title:Biology of the Cell
language:en
Short-container-title:Biol. Cell

Author:

Demangel Caroline¹²^ORCID,High Stephen³

Affiliation:

1. Immunobiology of Infection Unit; Institut Pasteur; Paris France

2. INSERM; U1221 Paris France

3. Division of Molecular and Cellular Function; School of Biological Sciences; Faculty of Biology; Medicine and Health; University of Manchester; Manchester M13 9PT UK

Funder

Institut Pasteur

Institut National de la Santé et de la Recherche Médicale

Fondation Raoul Follereau

Wellcome Trust

Publisher

Wiley

Subject

Cell Biology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/boc.201800030/fullpdf

Reference71 articles.

1. Mycolactone-mediated neurite degeneration and functional effects in cultured human and rat DRG neurons: mechanisms underlying hypoalgesia in Buruli ulcer;Anand;Mol. Pain,2016

2. Mycolactone subverts immunity by selectively blocking the Sec61 translocon;Baron;J. Exp. Med.,2016

3. Interferon-gamma is a crucial activator of early host immune defense against Mycobacterium ulcerans infection in mice;Bieri;PLoS Negl. Trop. Dis.,2016

4. The macrolide toxin mycolactone promotes bim-dependent apoptosis in Buruli ulcer through inhibition of mTOR;Bieri;ACS Chem. Biol.,2017

5. Mycolactone suppresses T cell responsiveness by altering both early signaling and posttranslational events;Boulkroun;J. Immunol.,2010

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