Senolytic treatment reduces cell senescence and necroptosis in Sod1 knockout mice that is associated with reduced inflammation and hepatocellular carcinoma-Reference-Cited by-同舟云学术

Senolytic treatment reduces cell senescence and necroptosis in Sod1 knockout mice that is associated with reduced inflammation and hepatocellular carcinoma

Published:2022-07-23 Issue:8 Volume:21 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Thadathil Nidheesh¹,Selvarani Ramasamy¹,Mohammed Sabira¹²,Nicklas Evan H.¹,Tran Albert L.¹,Kamal Maria³,Luo Wenyi³,Brown Jacob L.⁴⁵,Lawrence Marcus M.⁴⁶,Borowik Agnieszka K.⁴,Miller Benjamin F.⁴⁵⁷,Van Remmen Holly⁴⁵⁷^ORCID,Richardson Arlan¹²⁵⁷^ORCID,Deepa Sathyaseelan S.¹²⁷^ORCID

Affiliation:

1. Department of Biochemistry & Molecular Biology University of Oklahoma Health Sciences Center Oklahoma City Oklahoma USA

2. Stephenson Cancer Center University of Oklahoma Health Sciences Center Oklahoma City Oklahoma USA

3. Department of Pathology University of Oklahoma Health Sciences Center Oklahoma City Oklahoma USA

4. Aging & Metabolism Program, Oklahoma Medical Research Foundation Oklahoma City Oklahoma USA

5. Oklahoma City VA Medical Center Oklahoma City Oklahoma USA

6. Department of Kinesiology and Outdoor Recreation Southern Utah University Cedar City Utah USA

7. Center for Geroscience and Healthy Brain Aging University of Oklahoma Health Sciences Center Oklahoma City Oklahoma USA

Funder

National Institute on Aging

U.S. Department of Veterans Affairs

American Physiological Society

University of Oklahoma Health Sciences Center

National Institutes of Health

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13676

Reference64 articles.

1. Chemokine Signaling via the CXCR2 Receptor Reinforces Senescence

2. A complex secretory program orchestrated by the inflammasome controls paracrine senescence

3. Inflammatory and metabolic biomarkers and risk of liver and biliary tract cancer

4. Small Extracellular Vesicles Are Key Regulators of Non-cell Autonomous Intercellular Communication in Senescence via the Interferon Protein IFITM3

5. Age‐related dysregulation of inflammation and innate immunity: Lessons learned from rodent models;Brubaker A. L.;Aging and Disease,2011

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Sirtuin 3 is required for pro-inflammatory and pro-regenerative aspects of senescent cells;2024-08-22

2. Therapeutic strategies of targeting non-apoptotic regulated cell death (RCD) with small-molecule compounds in cancer;Acta Pharmaceutica Sinica B;2024-07

3. LILRB2 inhibition enhances radiation sensitivity in non-small cell lung cancer by attenuating radiation-induced senescence;Cancer Letters;2024-07

4. Cuproptosis: unveiling a new frontier in cancer biology and therapeutics;Cell Communication and Signaling;2024-05-01

5. MLKL overexpression leads to Ca2+ and metabolic dyshomeostasis in a neuronal cell model;Cell Calcium;2024-05