p38α‐MAPK‐deficient myeloid cells ameliorate symptoms and pathology of <scp>APP</scp> ‐transgenic Alzheimer's disease mice-Reference-Cited by-同舟云学术

p38α‐MAPK‐deficient myeloid cells ameliorate symptoms and pathology of APP ‐transgenic Alzheimer's disease mice

Published:2022-07-31 Issue:8 Volume:21 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Luo Qinghua¹²^ORCID,Schnöder Laura¹²,Hao Wenlin¹²,Litzenburger Kathrin¹²,Decker Yann¹²,Tomic Inge¹²,Menger Michael D.³,Liu Yang¹²^ORCID,Fassbender Klaus¹²

Affiliation:

1. Department of Neurology Saarland University Homburg Germany

2. German Institute for Dementia Prevention (DIDP) Saarland University Homburg Germany

3. Institute for Clinical and Experimental Surgery Saarland University Homburg Germany

Funder

Alzheimer Forschung Initiative

Deutsche Forschungsgemeinschaft

Universität des Saarlandes

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13679

Reference44 articles.

1. IL-17 triggers the onset of cognitive and synaptic deficits in early stages of Alzheimer’s disease

2. Microglia in Alzheimer's disease at single-cell level. Are there common patterns in humans and mice?

3. Microglia constitute a barrier that prevents neurotoxic protofibrillar Aβ42 hotspots around plaques

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