Differential profiles of gamma secretase Notch signalling in hidradenitis suppurativa: the need for genotype–endotype–phenotype analysis
Author:
Affiliation:
1. Department of Dermatology Liverpool Hospital Sydney Australia
2. University of New South Wales Sydney Australia
Publisher
Wiley
Subject
Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.19805
Reference7 articles.
1. Increased expression profile of NCSTN, Notch and PI3K/AKT3 in hidradenitis suppurativa
2. Impaired Notch signalling: the unifying mechanism explaining the pathogenesis of hidradenitis suppurativa (acne inversa)
3. γ-Secretase Gene Mutations in Familial Acne Inversa
4. Nicastrin mutations in familial acne inversa impact keratinocyte proliferation and differentiation through the Notch and phosphoinositide 3-kinase/AKT signalling pathways
5. Low Prevalence of GSC Gene Mutations in a Large Cohort of Predominantly Caucasian Patients with Hidradenitis Suppurativa
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1. Hidradenitis suppurativa: Detangling phenotypes and identifying common denominators;Journal of the European Academy of Dermatology and Venereology;2023-09-13
2. The genetic aspects of hidradenitis suppurativa;Clinics in Dermatology;2023-09
3. Obesity and hidradenitis suppurativa: targeting meta-inflammation for therapeutic gain;Clinical and Experimental Dermatology;2023-05-12
4. Variations in genetics, biology, and phenotype of cutaneous disorders in skin of color – Part I: Genetic, biologic, and structural differences in skin of color;Journal of the American Academy of Dermatology;2022-12
5. Identification of Biomarkers and Critical Evaluation of Biomarker Validation in Hidradenitis Suppurativa;JAMA Dermatology;2022-03-01
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