DLP1-dependent mitochondrial fragmentation and redistribution mediate prion-associated mitochondrial dysfunction and neuronal death-Reference-Cited by-同舟云学术

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DLP1-dependent mitochondrial fragmentation and redistribution mediate prion-associated mitochondrial dysfunction and neuronal death

Published:2017-11-22 Issue:1 Volume:17 Page:e12693
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Li Chaosi¹,Wang Di¹,Wu Wei¹,Yang Wei¹,Ali Shah Syed Zahid¹,Zhao Ying¹,Duan Yuhan¹,Wang Lu¹,Zhou Xiangmei¹,Zhao Deming¹,Yang Lifeng¹

Affiliation:

1. National Animal Transmissible Spongiform Encephalopathy Laboratory; State Key Laboratories for Agrobiotechnology; Key Laboratory of Animal Epidemiology and Zoonosis; College of Veterinary Medicine; Ministry of Agriculture; China Agricultural University; Beijing 100193 China

Funder

National Natural Science Foundation of China

Ministry of Agriculture of China

948 projects

Chinese Universities Scientific Fund

Publisher

Wiley

Subject

Cell Biology,Aging

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12693/fullpdf

Reference46 articles.

1. Dendritic and synaptic alterations of hippocampal pyramidal neurones in scrapie-infected mice;Belichenko;Neuropathol. Appl. Neurobiol.,2000

2. Mitochondrial fusion/fission dynamics in neurodegeneration and neuronal plasticity;Bertholet;Neurobiol. Dis.,2016

3. Early increase and late decrease of purkinje cell dendritic spine density in prion-infected organotypic mouse cerebellar cultures;Campeau;PLoS ONE,2013

4. Dysfunction of mitochondrial dynamics in the brains of scrapie-infected mice;Choi;Biochem. Biophys. Res. Comm.,2014

5. Synaptic degeneration is the primary neuropathological feature in prion disease - a preliminary-study;Clinton;NeuroReport,1993

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2. Assessment of the therapeutic potential of Hsp70 activator against prion diseases using in vitro and in vivo models;Frontiers in Cell and Developmental Biology;2024-07-22

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4. The PINK1/Parkin pathway of mitophagy exerts a protective effect during prion disease;PLOS ONE;2024-02-23

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